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Table 2 Arsenic exposure and carotid atherosclerosis.

From: GT-repeat polymorphism in the heme oxygenase-1 gene promoter and the risk of carotid atherosclerosis related to arsenic exposure

Average arsenic exposure, μg/L

Controls n (%)

Patients n (%)

Age- and gender-adjusted OR (95% CI)

Multivariate-adjusted OR (95% CI)

Lanyang cohort a

   ≤ 10

18 (7.0)

9 (3.6)

1.00 (referent)

1.00 (referent)

   10.1-50

10 (3.9)

9 (3.6)

2.54 (0.71-9.06)

2.58 (0.70-9.56)

   50.1-100

87 (34.0)

88 (35.2)

2.74 (1.13-6.64)*

2.98 (1.21-7.34)*

   100.1-300

79 (30.9)

81 (32.4)

2.82 (1.16-6.87)*

3.07 (1.23-7.65)*

   > 300

62 (24.2)

63 (25.2)

2.49 (1.01-6.15)*

2.62 (1.04-6.60)*

LMN cohort b

   ≤ 300

52 (31.7)

12 (10.3)

1.00 (referent)

1.00 (referent)

   300-750

65 (39.6)

56 (47.9)

2.03 (0.86-4.77)

1.93 (0.81-4.60)

   > 750

47 (28.7)

49 (41.9)

2.70 (1.12-6.47)*

2.78 (1.14-6.78)*

   Trend test

  

1.56 (1.04-2.34)*

1.61 (1.06-2.45)*

  1. OR: odds ratio; CI: confidence interval.
  2. a Adjusted for age, sex, logarithm triglyceride, total cholesterol, and hypertension history.
  3. b Adjusted for age, sex, hypertension history, and diabetes history.
  4. Age, triglyceride, and cholesterol were defined as continuous variables in the regression models.
  5. * p < 0.05.
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Journal of Biomedical Science

ISSN: 1423-0127