Fig. 1
From: Escape from IFN-γ-dependent immunosurveillance in tumorigenesis
Immune surveillance and escape from IFN-γ-dependent anticancer activity. CCL: CC chemokine ligands; CXCL: CXC chemokine ligands; CTL: cytotoxic T lymphocytes; CTLA: cytotoxic T-lymphocyte-associated protein; IDO: indoleamine-pyrrole 2,3-dioxygenase; IFN: interferon; IFNGR: IFN-γ receptors; IL: interleukin; IRF: IFN-regulating factors; JAK: Janus kinase; MDSC: myeloid-derived suppressor cell; MHC: major histocompatibility complex; NK: natural killer; PD-L1: programmed death ligand 1; PGE: prostaglandin E; SHP: src homology-2 containing phosphatase; SOCS: suppressors of cytokine signaling; STAT: signal transducer and activator of transcription; TAM: tumor-associated macrophage; TAP: transporter associated with antigen processing; TGF: transforming growth factor; TNF: tumor necrosis factor; TRAIL: TNF-related apoptosis-inducing ligand; Treg: regulatory T cell