Fig. 8
The motilities of the prc mutant strains with inactivated extracytoplasmic signaling pathways. (a) The motilities of the prc mutant strains of RS218 with a blocked σE, RcsCDB, CpxA-CpxR, QseB-QseC, or EnvZ-OmpR system. degS, rcsB, cpxR, qseB, and ompR deletions were introduced into Δprc-RS218 to block the σE, RcsCDB, CpxA-CpxR, QseB-QseC, and EnvZ-OmpR signaling pathways, respectively. (b) The motility of the ΔdegsΔprc-RS218 strain complemented with degS in the chromosomal lacZ locus. (c) The motility of the ΔrcsBΔprc-RS218 strain complemented with rcsB in the chromosomal lacZ locus. (d and e) The motilities of the prc mutant strains of CFT073 with an inactivated σE (d) or RcsCDB (e) system