Fig. 11From: Targeting influenza A virus by splicing inhibitor herboxidiene reveals the importance of subtype-specific signatures around splice sitesLow expression of H3N2 M2 can efficiently activate LC3 cleavage. Western blot showing expression of viral proteins using specific antibodies. A549 cells were infected by H1N1 or H3N2 at MOI 0.001, and cell lysates were collected at 12, 24, and 36Â h post-infection. Top panel: H1N1 wild type (WT) (M/55C) and mutant (Mut) (M/C55T). Bottom panel: H3N2 WT (M/55T) and Mut (M/T55C). Densitometric analysis was performed using ImageJ softwareBack to article page