Fig. 4
From: Targeting NLRP3 signaling reduces myocarditis-induced arrhythmogenesis and cardiac remodeling
Estimates of INa-Late, ICa-L, and NCX current in rat RVOTs. A Tracings and I–V relationship of NCX current in the RVOT cardiomyocytes of the control (healthy rats; n = 10), experimental (rats with myocarditis; n = 10), and treatment (MCC950-treated rats with myocarditis; n = 9) groups. B Tracings and I–V relationship of ICa-L in the RVOT cardiomyocytes of the control (n = 14), experimental (n = 16), and treatment (n = 12) groups C Current tracings and average data of INa-Late in the RVOT cardiomyocytes of the control (n = 12), experimental (n = 12), and treatment (n = 8) groups. INa-Late, late Na+ current; ICa-L, L-type Ca2+ current; NCX, Na+–Ca2+ exchanger; RVOT, right ventricle outflow tract