Fig. 9From: Attenuating mitochondrial dysfunction and morphological disruption with PT320 delays dopamine degeneration in MitoPark miceIn summary, our findings suggest that PT320 regulates mitochondrial morphology by promoting Opa1 expression and inhibiting Fis1 expression during the early stage. Additionally, we observed that Opa1 has an influence on the release of cytochrome c, which is an upstream factor in apoptosis, by regulating the structure of mitochondrial cristaeBack to article page